An increasingly utilized front-line therapy, particularly vital when no-reflow causes severe systemic hypotension that contraindicates the use of vasodilatory agents like adenosine or nitroprusside. Administered in 50 to 200 µg intracoronary boluses, epinephrine's beta-2 agonism drives potent microvascular dilation, while its simultaneous beta-1 effects provide essential inotropic support to the stunned myocardium.
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